Non-Obstructive Coronary Artery Disease due to COVID-19 Infection: St-Segment Elevation due to Endothelial Dysfunction

The ongoing inflammatory process in COVID infection is reported to cause acute cardiac injury either due direct cytotoxicity or oxidative stress myocytes. Hypercoagulability and endothelial dysfunction are the two main events resulting transient thrombotic process. Objective: To assess coronary artery occlusion infected patients presenting with ST-elevation myocardial injury. Methods: A descriptive case series study was conducted on 52 infarction along infection. Data collected regarding demographic profile, Troponin levels, PCR, changes ECG, Echocardiographic findings arteries were assessed angiographically. analyzed using SPSS software various percentages calculated. Results: Of all studied, 46 (88%) tested positive for -19 RT-PCR done via nasopharyngeal swab. Other 6 negative initially but they had their CXR HRCT suggestive of interstitial pneumonia. All age group 30-60 years 32 (62%) being males 20 females (38%). None previously any history angina infarction. Inflammatory markers raised about 43 (82.7%). ECG showed ST segment elevation anterior leads 35 (67%) inferior 9 (17%) lateral 2 (3.8%). Qualitative I test patients, whereas, 41 (78%) Quantitative high probability range. About 50 (96%) Ejection Fraction greater than 45% normal segmental wall motion analysis. be recanalized 40 (76%) mild-moderate disease single vessels present rest 12 patients. shown culprit severe clot burden angiograms. Conclusions: Acute Cardiac asymptomatic may related pro-thrombotic state created by COVID-19 mimicking raising levels indicated D-Dimers CRP level. auto-recanalization echocardiographic findings, good prognostic factors post rehabilitation.